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--- Core Faculty    


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N O P Q R S T U V W X Y Z 
  Last Name First Name
AlexanderGene
AllenJohn
ArkowitzHarold
BarnesCarol
BechtelRobert
BeckConnie
BeckerJudith
BedfordFelice
BeverThomas
BootzinRichard
DanielTerry
FellousJean-Marc
FigueredoAurelio
FontaineReid
ForsterKen
FrybergStephanie
GerkenLouAnn
GliskyElizabeth
GomezRebecca
GreenbergJeff
JacobsW. Jake
KaszniakAlfred
LunsfordLaura
MehlMatthias
NadelLynn
NicolJanet
PetersonMary
Piattelli-PalmariniMassimo
RohrbaughMichael
RyanLee
SalesBruce
SanfeyAlan
SbarraDavid
ScheresAnouk
SchwartzGary
ShohamVarda
StoneJeff
Tel: 621-7449
Office: 513
Building: Psychology
nadel@u.arizona.edu
Personal Homepage: None
 

Nadel, Lynn


Regents' Professor

Program: Cognition and Neural Systems
Year of affiliation: 1985
Year of doctor degree: 1967
School: McGill University, Montreal

       My research is concerned with the role of the hippocampal formation in learning and memory, and focuses on its particular role in spatial cognition. My earlier work demonstrated that this brain structure was critical for place learning, and is the core of a brain system responsible for creating neural cognitive maps of environments that an animal has explored and must remember. My current work focuses on five aspects of hippocampal function in spatial cognition: (1)the implications of the fact that this cognitive system matures postnatally, in most species only after some months; (2)the computational mechanisms by which the hippocampus actually accomplishes the sotrage and use of cognitive maps; (3)the way in which the hippocampus interacts with the neocortex in the long-term stabilization of memory; (4)gender differences in spatial cognition; and (5) the way in which this cognitive mapping function in humans is expressed in both nonverbal spatial mapping systems and verbal semantic mapping systems. This work involves the study of both animals and humans, as well as computer simulation of hippocampal circuitries and their interaction with neocortical systems. It employs standard behavioral techniques in combination with either experimentally-induced brain damage, or developmentally produced disorders of hippocampal function (as in Down syndrome).

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